InhiBET™ BET Inhibitor Platform

VYNE is developing novel therapies for immuno-inflammatory (I&I) diseases that target bromo-domain and extra-terminal domain proteins (BET).

InhiBET™ BET Inhibitor Platform:

BETs are a family of proteins characterized by two bromo-domain subunits (BD1 and BD2) plus an extra-terminal domain (EXT). BET proteins are believed to play a key role in the regulation of inflammatory and oncogenic genes involved in several diseases.

VYNE’s BET inhibitors (BETi’s) are based on its proprietary InhiBETTM platform and small molecule library.


The BET Protein: BD1 and BD2 are bromodomains and EXT is an extra-terminal domain

InhiBET™ BET Inhibitor Programs:

VYN201: Locally administered Pan-BD BET inhibitor

Target Markets:

Designed to address diseases involving multiple, diverse inflammatory cell signaling pathways with low systemic exposure

  • Lead indication – Vitiligo
  • Other indications benefiting from local application and “soft drug” approach
Broad activity:
  • Binds to BD1 and BD2 domains
Status / Targeted Near Term Milestones::
  • Phase 1b completed
  • Q2 2024: P2b initiated


BET Protein

VYN202: Oral BD2-selective BET inhibitor

VYN202 has been designed to achieve class-leading selectivity (BD2 vs. BD1), maximum potency versus BD2 and optimal oral bioavailability. Maximizing on-target potency vs. BD2 and minimizing affinity to BD1 may be the key to optimizing the benefit/risk profile of BET inhibitors for autoimmune diseases.

Target Markets:
  • Moderate-to-severe plaque psoriasis
  • Moderate-to-severe rheumatoid arthritis
Focused activity:
  • VYN202 is believed to be the most potent and BD2-selective BET Inhibitor in development1 which is designed to improve efficacy and tolerability
Status / Targeted Near Term Milestones:
  • Q2 2024: P1a SAD/MAD initiated

1 Based on readily available public information such as clintrials.gov, academic publications and corporate websites/presentations.